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Moreover, ivacaftor treatment of monocyte-derived macrophages from cystic fibrosis patients increased the expression of CFTR, partially restore macrophage polarization and bacteria killing, and reduced the secretion of pro-inflammatory cytokines by these cells (Barnaby et al., 2018)

In accord with this view, TRPM2 knockout was shown to inhibit necrotic cell death induced by H 2 O 2 or Zn 2+ in mouse macrophages (Mortadza et al., 2017), by FPP in mouse mastrocytoma cells (Zong et al., 2022a)